This study describes the interactions between two different pro-inflammatory factors produced by bacteria, lipopolysaccharide (LPS) from Gram-negative bacteria and the peptide BacSp222 produced by a ...

This study investigates neuro-inflammation and amyloidogenesis and memory impairment following the systematic inflammation generated by lipopolysaccharide.

Cite this: Neuro-Inflammation Induced By Lipopolysaccharide Causes Cognitive Impairment Through Enhancement Of Beta-Amyloid Generation - Medscape - Aug 29, 2008.

Detailed price information for Inmed Pharmaceuticals Inc (INM-Q) from The Globe and Mail including charting and trades.

Immune activation in the body is well studied; however, much less is known about how peripheral inflammation changes brain chemistry. Because depression and inflammation are close comorbidities, ...

LptC regulates the LPS entry gate in LptB2FG, likely through a dynamic behavior of its transmembrane helix, while the β-jellyroll domains are tethered in the periplasm.

Lipopolysaccharide (LPS), the cell wall component of Gram-negative bacteria, can damage the barrier function of BBB and further promote the occurrence and development of sepsis-associated ...

p204, a murine member of an interferon-inducible p200 family, was reported to recognize intracellular viral and bacterial DNAs, however, its role in the innate immunity in vivo remains unknown due to ...

Lipopolysaccharide Detection by the Innate Immune System Early detection of pathogen-associated molecular patterns (PAMPs) universally shared by Gram-negative bacteria is a crucial element for the ...

image image Glucocorticoids limit the effects of LPS in vivo at two critical control points, namely at the level of macrophages by controlling TNF production, and at the level of intestinal ...

Epidemiological studies have reported a link between serum LBP (lipopolysaccharide‐binding protein) levels and lifestyle‐related diseases. However, there have been no longitudinal studies ...