Alterations in oncogenes and tumor suppressor genes can drive the pathogenesis of DLBCL. 8, 9 Two such oncogenes are MYC and BCL2, key regulators of cellular proliferation and apoptosis, respectively.

Vitamin C helps reactivate growth genes in your skin through DNA demethylation, promoting cell renewal, thickness, and a ...

Perhaps best known as a immune-system boost or antidote to scurvy, vitamin C has been found to rejuvenate aging skin by ...

Using a 3D human skin model, they showed that C boosts thickness in the epidermal skin layer by activating genes linked to cell growth.

Researchers at Karolinska Institutet have discovered a previously unknown molecule that may explain why people with type 2 diabetes often suffer from muscle weakness and muscle loss - a condition that ...

It also caused a drop in the activity of genes in the MYC pathway. This suggests that without the epigenetic function of SETD1B, MYC cannot remain fully active, weakening the ability of the cancer ...

The tumor suppressor p53 is a transcription factor that controls the expression of hundreds of genes. Emerging evidence suggests that the p53-induced RNA-binding protein ZMAT3 is a key splicing ...

Background Chronic inflammation and elevated reactive oxygen species are key contributors to hepatocellular carcinoma (HCC) ...

Bose, Smith-Bolton, and their coauthors hypothesized that particular genes are responsible for making sure that regeneration ends with the right number and types of cells. One of the genes that ...

The mRNA metabolism passively shapes the levels of an mRNA modification called m6A within a steady-state cell and upon stress.